Study Finds People Given Basic Income Are Likely To Keep Working

Universal Basic Income (UBI) is a social concept in which every person within a system receive a regular, standard, basic income regardless of employment, background or other external factors. Some criticize UBI, saying that people would stop working with a basic income. However, a recent study demonstrated that people receiving UBI would likely keep their jobs and even take on more part-time work.

The study examines the impact of the Alaska Permanent Fund, a $61-billion communal resource backed by oil, which has been running for more than 35 years. It is currently the closest thing to a UBI in the U.S. Researchers at the University of Chicago Harris School of Public Policy analyzed the economic effect of the annual cash payments made to Alaskan residents, which have recently totaled around $2,000 per person.

The researchers found that not only did employment not decrease, but the number of people in part-time work actually increased by a significant 17 percent. And, while overall employment was reduced in fields like manufacturing and oil, it remained steady in fields like construction, education, and healthcare.

The researchers concluded in a working paper published in February in the National Bureau of Economic Research that “Our results suggest that a universal and permanent cash transfer does not significantly decrease aggregate employment.”

As automation increasingly threatens human-held occupationspoliticians and experts alike are exploring how realistic a solution UBI could be to this job loss. Not all agree that UBI is the right fix for automation-linked job loss, but this researchcould spark further investigationto see if it could be a good fit for the U.S.

Once it is further reviewed, this data could potentially convince those on the fence that UBI is, at the very least, worth investigating with small, U.S.-based trials. It at least opens up the discussion by suggesting that, even when people receive supplemental income from the government, their drive to work doesn’t simply dissipate. Instead, the fact that people have more money to spend could actually create jobs.

However, Alaska’s program is not a perfect representation of UBI. $2,000 a year couldn’t be viewed as an income replacement, even a part-time income replacement. So, while this study demonstrates that employment trends might not be what some projected, it is difficult to know exactly what to expect because larger payments might cause people to respond differently.

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Study Finds Numerous Life-Threatening Injuries, Hospitalizations & Deaths After Gardasil (HPV) Vaccine



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A new study published in Clinical Rheumatology exposes how vaccine manufacturers used phony placebos in clinical trials to conceal a wide range of devastating risks associated with HPV vaccines. Instead of using genuine inert placebos and comparing health impacts over a number of years, as is required for most new drug approvals, Merck and GlaxoSmithKline spiked their placebos with a neurotoxic aluminum adjuvant and cut observation periods to a matter of months.

Researchers from Mexico’s National Institute of Cardiology pored over 28 studies published through January 2017—16 randomized trials and 12 post-marketing case series—pertaining to the three human papillomavirus (HPV) vaccines currently on the market globally. In their July 2017 peer-reviewed report, the authors, Manuel Martínez-Lavin and Luis Amezcua-Guerra, uncovered evidence of numerous adverse events, including life-threatening injuries, permanent disabilities, hospitalizations and deaths, reported after vaccination with GlaxoSmithKline’s bivalent Cervarix vaccine and Merck’s quadrivalent or nine-valent HPV vaccines (Gardasil and Gardasil 9). Pharmaceutical company scientists routinely dismissed, minimized or concealed those injuries using statistical gimmicks and invalid comparisonsdesigned to diminish their relative significance.

Scientific researchers view double-blind placebo trials as the gold standard for testing new drugs. To minimize bias, investigators randomly assign patients to either a “treatment” group or a “control” (placebo) group and then compare health outcomes. The standard practice is to compare a new drug against a “pharmacologically inert” placebo. To minimize opportunities for bias, neither patients nor researchers know which individuals received the drug and which the placebo. However, in clinical trials of the various HPV vaccines, pharmaceutical researchers avoided this kind of rigor and instead employed sleight-of-hand flimflams to mask the seriousness of vaccine injuries.

Of the 16 HPV vaccine randomized trials, only two used an inert saline placebo. Ten of the sixteen compared the HPV vaccine against a neurotoxic aluminum adjuvant, and four trials used an already-approved aluminum-containing vaccine as the comparison. One does not have to be a scientist to understand that using aluminum-containing placebos is likely to muddy the comparison between the treatment and control groups. Critics of the HPV vaccine have pointed to the aluminum adjuvant as the most likely cause of adverse reactions, and some researchers have questioned the safety of using aluminum adjuvants in vaccines at all, due to their probable role as a contributor to chronic illness. The aluminum-containing placebos appeared to provoke numerous adverse reactions among the presumably unwitting patients who received them, allowing the pharma researchers to mask the cascade of similar adverse reactions among the groups that received the vaccines. Although both placebo and study groups suffered numerous adverse events in these studies, there were minimal differences between the two groups. The similar adverse outcomes in both groups allowed industry researchers and government regulators to claim that the vaccines were perfectly safe, despite manifold disturbing reactions. The Mexican researchers’ meta-review confirms the difficulty of ascertaining vaccine-attributable differences from this mess; the researchers identified only a few indications of “significantly increased systemic adverse events in the HPV vaccine group vs. the control group” across the 16 pre-licensure trials.

The HPV promoters found it more difficult to employ deceptive devices in the 12 post-marketing safety reviews, and the Mexican authors summarize some of the more noteworthy findings. In Spain, they found a ten-fold higher incidence of vaccine-related adverse events following HPV vaccination compared to “other types of vaccines.” In Canada, they found an astonishing one in ten rate of hospital emergency department visits among HPV-vaccinated individuals “within 42 days after immunization.” Still, the industry researchers did what they could to minimize these injuries. The Mexican reviewers criticize the authors of the various post-marketing studies for failing to ask essential questions, to evaluate the many serious adverse events, or to elaborate on their often-troubling findings.

Abbreviated Trial Times

Typically, FDA requires drug companies seeking approval for a new drug to observe health outcomes in both the placebo and study groups for 4-5 years. Vaccine manufacturers take advantage of FDA regulatory loopholes that allow fast-tracking of vaccines and cut that period down to a few weeks or even a few days. This means that injuries that manifest, or are diagnosed, later in life—most neurodevelopmental disorders, for example—will escape attention entirely.

Further Smokescreens

Martínez-Lavin and Amezcua-Guerra point to clinical trial data posted on the FDA webpagefor the quadrivalent Gardasil vaccine approved in 2006. Those clinical trials deployed a panoply of the kind of cunning deceptions used by industry and government researchers. Unlike many of the other HPV vaccine clinical trials, these clinical studies employed a true saline placebo.

Across the Gardasil clinical studies, a group of 15,706 females ages 9-45 and males ages 9-26 received the quadrivalent Gardasil vaccine. A control group of 594 individuals received an inert saline placebo. The industry researchers never explain the tiny relative size of the saline placebo group; it’s noteworthy that small size would have the effect of keeping unwanted signals weak. But a second control group of 13,023 received a so-called “spiked” placebo loaded with an aluminum adjuvant (amorphous aluminum hydroxyphosphate sulfate or AAHS). The large size of this “spiked placebo” group suggests that the decision to keep the saline placebo group small was strategic.

Putting aside the thorny ethical question of whether study participants were told that they were being injected with a neurotoxin with probable associations with Alzheimer’s, dementia and other forms of brain disease, the inclusion of both saline and aluminum placebos provided these researchers a chance to do some genuine science. But the FDA webpage shows the troubling gimmick that was then employed by the FDA and Merck, which seems deliberately designed to blur datasets in order to mask adverse effects during the clinical trials. The table showing relatively minor injection-site adverse reactions—one to five days post-vaccination—displays three distinct columns for the three groups: Gardasil recipients, the aluminum “placebo” recipients, and saline placebo recipients (see table below). In the table, “Intergroup differences are obvious,” in the words of the Mexican researchers. For example, roughly three and a half times more girls/women experienced injection site swelling in the Gardasil group compared to the saline group (25.4% vs. 7.3%). In fact, by all five measures, both the Gardasil recipients and the aluminum placebo recipients fared two to three times worse than the saline recipients.

When it came time for Merck to report on the occurrence of more serious reactions, “Systemic Adverse Reactions” and “Systemic Autoimmune Disorders,” for example, the company scientists switched to a very different format. In these tables, the third column that reported results for the saline placebo recipients disappears. Instead, Merck combined the groups receiving the spiked aluminum placebo into a single column with the group receiving the genuine saline placebo (see example below). The merger of the two control groups makes it impossible to compare results for Gardasil versus the saline placebo or the aluminum placebo versus the saline placebo. In this way, Merck’s researchers obliterated any hope of creating a meaningful safety comparison.

Risks and Benefits

Given aluminum’s known neurotoxicity and its association with debilitating autoimmune conditions, it is unsurprising that there are no observable differences between the Gardasil and AAHS/saline groups. But, despite the researchers’ efforts to paper over adverse effects, they were not able to conceal the devastating health injuries to their human guinea pigs. The bottom line of these trials reveals a shocking truth: An alarming 2.3% of both their study and control groups had indicators of autoimmune diseases! These data are even more alarming when one considers that the observation period was curtailed after only six months. With this level of risk, it would seem that no loving parents would allow their daughter to receive this vaccine—particularly given the comparatively low risk posed by HPV in countries with appropriate cervical cancer screening tests. Even in countries such as India, where cervical cancer mortality is high due to late detection, leading Indian physicians argue that comprehensive screening should be the country’s top priority rather than the “panacea” of HPV vaccination.

Consider the math: According to the National Institutes of Health (NIH), an estimated 2.4 women per 100,000 die of cervical cancer in the US each year. On the other hand, the FDA’s Table 2 (above) shows that 2.3 per 100 girls and women developed an “incident condition potentially indicative of a systemic autoimmune disorder” after enrolling in the Gardasil clinical trial. It is difficult to understand how any rational regulator could allow more than two in 100 girls to run the risk of acquiring a lifelong autoimmune disorder, particularly when Pap smears are already doing an effective job of identifying cervical abnormalities. The NIH notes that the incidence and death rates for cervical cancer in the US declined by more than 60% after introducing Pap smear screening.

Martínez-Lavin and Amezcua-Guerra make their own effort to illustrate the zany risk-benefit ratios associated with these vaccines when discussing the results of one of the 16 clinical trials. That study compared approximately 14,000 women who received either Gardasil 9 or the original quadrivalent Gardasil. Based on the numerical outcomes of that study, the Mexican researchers calculated the likelihood of being actually “helped or harmed by the 9-valent HPV vaccine.” Their “worrisome” finding is that the “number needed to harm” is just 140, whereas 1757 women would need to receive the vaccine for a single one of them to enjoy its projected benefits.

Implications for Aluminum Adjuvants

Merck found that astronomical casualty counts were equal among both Gardasil and aluminum “placebo” recipients. The inescapable implication is that aluminum adjuvants may be a principal culprit in the flood of injuries reported for the various HPV vaccines. This conclusion, if true, requires reevaluation of the use of aluminum adjuvants in several other vaccines, including some given to infants. Aluminum adjuvant levels have mushroomed since the 2003 removal of thimerosal from three pediatric vaccines. The following chart, prepared by Dr. Sherri Tenpenny, illustrates the stunning amount of aluminum in vaccines.

Multiple peer-reviewed studies have connected aluminum exposures to a range of autoimmune and neurological disorders, including dementia and Alzheimer’s disease, that have become epidemic coterminous with these aluminum exposures. A review in the European Journal of Clinical Nutrition warns of dangerous accumulation of aluminum in the brain when, as in the case of vaccination, “protective gastrointestinal mechanisms are bypassed.” It’s time to go back to the drawing board on HPV vaccines and aluminum adjuvants. More importantly, FDA needs to start requiring the same rigorous pre-licensing safety testing for vaccines that it has long required for other drugs. All existing vaccines, particularly those containing aluminum, should be safety-reviewed according to these more stringent standards.

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Groundbreaking Study Shows Shielding EMF Improves Autoimmune Disease



Concerns about electromagnetic fields (EMF) are branded pseudoscientific conspiracy theories and relegated to the realm of tin-hat wearing quackery. However, a recent publication in the peer-reviewed journal Immunologic Research entitled “Electrosmog and Autoimmune Disease,” sheds new light on the validity of concerns about this so-called electrosmog with which we are constantly inundated.

Although we encounter natural microwave electromagnetic radiation in the form of cosmic radiation from outer space, the aurora borealis, and thunderstorms, the vast majority of electrosmog that we encounter is largely manmade (1). These atmospheric phenomena, however, emit electromagnetic radiation at lower radio frequencies and are negligibly weak in comparison to manmade sources, which have increased exponentially due to the emergence of television, cellular phone technologies, and WiFI, all of which utilize microwave frequency bands (1).

According to researchers Marshall and Heil (2017), for instance, “The recent release of WiGig and anti-collision vehicle radars in the 60 GHz region embody a 1000-fold increase in frequency, and photon energy, over the exposures mankind experienced up until the 1950s” (1).

How Electrosmog Interfaces with the Bioelectromagnetic Body

It is intuitive that electrosmog would interact with human biology, since human physiology operates in part via electromagnetic fields. Apart from physical information superhighways such as the blood, nervous, and lymphatic systems, the body uses electromagnetic forms of energy transmission and communication which are several orders of magnitude faster than chemical diffusion (2).

Called biophotonic emission (BPE), these quanta of electromagnetic energy have a visibility one thousand times lower than the sensitivity of our naked eye and are quintessential to cellular metabolism and to the powering of our energy-intensive nervous and immune systems (3). Harbored within our genetic material, biophotonsserve as a mode of instantaneous communication from one body part to another and to the extraneous world (4) and their emission is influenced by our global state of health (5). Research even suggests that mental intention and the fabric of our consciousnessis mediated by these quantum of light, which operate as highly coherent frequencies and generate an ordered flux of photons (4).

Thus, both the stuff of consciousness and the functioning of our cellular energetics is premised upon electromagnetism, which may be susceptible to distortion by electrosmog. Curtis and Hurtak describe the electromagnetic body as both “an entire body distinct from the chemical body that interpenetrates it” and “a light circulatory system operating on an energetic level in a markedly different manner from that of its molecular counterparts” (2). That there is “an incredible amount of activity at levels of magnification or scale that span more than two-thirds of the 73 known octaves of the electromagnetic spectrum” (6) in the human body is emblematic of our vulnerability to electromagnetic disturbances.

Potential Immune Disturbances due to Electrosmog Exposure

Although current public health laws are predicated on effects of short-term exposure, research suggests that dosage and repetitive exposures likely influence health risk of electrosmog (7). Two thirds of studies examined report ecological effects of electromagnetic radiation, and researchers state that, “current evidence indicates that chronic exposure to electromagnetic radiation, at levels that are found in the environment, may particularly affect the immune, nervous, cardiovascular and reproductive systems” (7).

Although the conventional mantra is that no harm is incurred from low-energy radio waves, low-level exposures to ionizing radiation are known to manifest profound effects upon human physiology (1). Ionizing radiation exposure, which occurs secondary to nuclear energy accidents, for example, produces immunosuppression, so much so that some scientists have even suggested radon exposure as a therapeutic treatment for rheumatoid arthritis due to its inhibition of inflammatory immune messengers such as the adipokine visfatin (8).

There is, however, often a substantial lag time between exposure and the materialization of symptomatology (1). The detriment to immune defense “often does not become apparent until the body catastrophically fails to overcome an acute challenge” (1). In addition, new science is overturning the previous assumption that immunosuppressive effects are exclusive to ionizing radiation exposure.

A research group headed by Lushinov, for example, found that repeated exposures to low-level non-ionizing electromagnetic radiation impaired the immune response in mice, negatively influencing immunogenesis, or the ability of the immune response to respond to an immune-provocating antigenic substance (9). The exposure to low-intensity electromagnetic radiation negatively influenced thymic and splenic cellularity, causing a statistically significant decrease in the immune cells generated by these lymphoid organs (9). The immunocompetence of the Aegean wall lizard was also significantly reduced upon daily exposure to radiofrequency resembling the amount of electrosmog emitted from cordless phones (10).

Moreover, Gapeev and colleagues (2006) elucidated that exposure to low-intensity non-ionizing electromagnetic waves exerted equivalent immunosuppressive effects to a single dose of the nonsteroidal anti-inflammatory drug (NSAID) diclofenac (11). In another experiment, exposure to low-intensity electromagnetic radiation reduce the footpad edema and local hyperthermia, also known as swelling and heat, that accompanied injection of zymosan, an agent that induces acute inflammation (12). This constitutes evidence that electrosmog exposure may impair the normal immune response to potential threats.

Human Proteins are Responsive to Electromagnetic Waves

Biomolecules, which are constantly undergoing molecular collisions and interacting on the scale of picoseconds, are subject to forces exerted by incident electromagnetic fields (1). According to researchers Marshall and Heil, “It seems likely that signals a million times lower than those currently being used in research may be sufficient to elicit a tangible change in human biology” (1).

Induction of Stress Proteins

Electrosmog at both an extremely low-frequency (ELF) or in the radio frequency (RF) range has been found to stimulate a cellular stress response, leading to expression of stress response genes including heat shock protein 70 (HSP70) (13). As a consequence, there is increased production of highly conserved stress proteins, which serve as chaperones by refolding and repairing damaged proteins (13). Heat shock proteins have likewise been observed to up-regulate an immune response, “transferring antigenic peptides to the class I and class II molecules of the major histocompatibility complexes” as well as increasing activity of a class of immune cells which perpetuate an immune reaction, such as macrophages and dendritic cells (14).

Aberrant Anti-Microbial Response

In addition, the function of another human protein, lysozyme, has been shown to be disrupted by electromagnetic radiation (15). Also called muramidase, lysozyme is an antimicrobial enzyme liberated from cytoplasmic granules of immune cells such as granulocytes and macrophages (16). Contained in human secretions such as mucus, tears, saliva, and breast milk, this bacteriolytic element degrades glycosidic bonds in peptidoglycan, a molecule prominent in the cell walls of gram-positive bacteria (17).

Lysozyme is a major contributor to bactericidal activity, facilitating elimination of inhaled airborne microorganisms to prevent their colonization in the respiratory passages, which would interfere with sterile gas exchange (17). Studies have indicated that depletion of lysozyme reduces bacteria-killing ability of human airway sections by approximately fifty percent (18). Animal studies also highlight how lysozyme is especially important in host pulmonary defense, since, “Increased concentration of lysozyme in the airspaces of transgenic mice enhanced bacterial killing whereas lysozyme deficiency resulted in increased bacterial burden and morbidity” (17).

Turton and colleagues (2014) published a study in Nature Communications showing that non-ionizing terahertz electromagnetic radiation altered the binding of lysolyme to its ligand, triacetylchitotriose, which in turn would affect the biological function of lysozyme (15). Although this represents a much higher frequency than normal background electrosmog, the implications are that human immune defenses against pathogen invasion and virulence may be adversely affected due to repeated and cumulative exposures to electrosmog (15).

Derangements in Vitamin D Pathways

Research shows that Vitamin D Receptor (VDR) pathways are susceptible to interference by electrosmog (1). Functionality of the vitamin D receptor, a transcription factor that translocates to the nucleus and influences gene expression when bound to vitamin D, is fundamental for immunomodulation. The cascade of effects that occur upon vitamin D binding to its receptor reinforce gut barrier integrity, establish oral tolerance, and suppress autoimmune responses by enabling the immune system to differentiate self from non-self.

According to researchers, the shape of the VDR molecule transforms with electrosmog exposure within the frequency range of WiFi routers: “Groups of hundreds of atoms which form the helical “backbone” of the VDR…shift together at the lower frequencies present in electrosmog” (1). Sophisticated molecular dynamics software, which illustrates the lock-and-key interaction between the vitamin D receptor and its native ligand, 1,25-dihydroxyvitamin-D (1,25-D), have shown that so-called Lorentz forces act upon charged oxygen atoms in carboxyl groups of the vitamin D receptor (1). These Lorentz forces may either promote or hinder activation of the vitamin D receptor, depending on both the frequency of the “molecular interactions, and that of the impinging electromagnetic waves” (1).

Electrosmog Affects Human Brain Activity and Behavior

As far back as 1987, Bise published a pilot study wherein electrosmog exposure at levels dramatically lower than that observed in urban areas elicited transient changes in human brain waves and behavior (19). He reports, “Constructive and destructive interference patterns from standing waves within the skull possibly interact with the bioelectric generators in the brain, since electroencephalogram wave amplitudes and frequencies increased or decreased respectively at different radio wavelengths” (19).

What’s more, the literature reveals that neuroimaging and electroencephalography studies demonstrate enhanced cortical excitability with EMF exposure, particularly in the front-temporal regions, which is paradoxically correlated with faster reaction times, but may also interfere with sleep (20).

Alarmingly, the patterns observed in human electroencephalograms (EEG) was altered by wave amplitudes as low as -100 dBm (19). Bise was able to induce an immediate frontal headache at a level of -60 dBm (19). Unfortunately, barring use of a Faraday cage, these experiments are impossible to replicate since electrosmog background levels in cities are now 100,000 times stronger at -50 dBm (19).

Silver-Threaded EMF-Blocking Caps Improve Autoimmune Disease

In a recent case series, patients wore shielding clothing and tenting consisting of silver-coated polyester threads interspersed with bamboo fibers that were partially capable of blocking penetration of microwave electrosmog (1). Due to anecdotal testimonies of improvement, researchers decided to distribute standardized garments that would shield the brain and brain stem in order to systematically analyze the results (1).

In this study, 64 patients with assorted autoimmune diagnoses such as systemic lupus erythematosus (SLE)rheumatoid arthritis (RA)multiple sclerosis (MS)Sjogren’s syndrome, and celiac disease, many of whom were disabled and house-bound, were recruited (1). Subjects wore the silver-threaded cap for four hours at night and for four hours during the day, and patient-reported outcomes were collected (1). Impressively, 90% of patients indicated a “definite” or “strong” change in their symptomatology, which is at variance with the 3% of the population that is estimated to be sensitive to electrosmog (1).

Some researchers have attributed this so-called electro-hypersensitivity (EHS) or idiopathic environmental intolerance (IEI) to the nocebo effect. However, Dieudonné explores the possibility of a psychosomatic mechanism in the journal Bioelectromagnetics, and concludes, “Overall, symptoms appear before subjects start questioning effects of EMF on their health, which is not consistent with the hypothesis that IEI-EMF originates from nocebo responses to perceived EMF” (21).

In this groundbreaking study, it is also telling that the researchers found the therapeutic efficacy of the silver-coated caps to be so theoretically plausible that they decided the idea of using a control group was unethical. These authors concluded that autoimmune patients exhibit a pronounced susceptibility to electrosmog at levels normally encountered in home and occupational environments, and hypothesized that the exposure may be contributing to their disease etiology (1).

Electrosmog and Mitochondrial Dysfunction

Because electric fields result from voltage differences, whereas magnetic fields from the flow of electric current, EMFs may be capable of disrupting the finely orchestrated proton gradient and flow of electrons within the inner mitochondrial membrane upon which the process of oxidative phosphorylation is contingent (13). Oxygen-dependent aerobic respiration, which relies upon oxidative phosphorylation, is the process that drives production of the cellular energy currency adenosine triphosphate (ATP) in our cellular energy factories, the mitochondria.

These organelles are fundamental to every energy-dependent process in the body but especially quintessential for the energy-demanding nervous system. Thus, EMF-mediated changes in mitochondrial function may affect cognition and even perpetuate development of neurodegenerative diseases such as Alzheimer’s and Parkinson’s in which mitochondrial dysfunction has been demonstrated. In fact, EMF-induced disruption of mitochondria may play a role in many diseases in which mitochondrial collapse is implicated, including psychiatric disordersautoimmune diseasesmigraine headachesataxiastrokediabetes, heart disease, neuropathic pain, chronic fatigue syndrome, fibromyalgia, and liver disease (22, 23).

It has also been proposed that EMFs can interact directly with electrons in DNA, so it is not a stretch that EMFs could interact with the electron transport chain (ETC) in mitochondria (24). This concept is supported by a study where pulsed electromagnetic radiation (EMR) resulted in alterations in the ETC, leading to adverse metabolic changes, cellular hypoxia, and increased generation of oxidative stress inducing free radicals such as the superoxide anion (25).

Electrosmog and Cancer

Although the undoubtedly industry-influenced mainstream consensus is that EMFs do not play a role in the development of childhood cancers, “Kheifets and Shimkhada [2005] stated that epidemiologic studies of ELF-EMFs and childhood leukemia are difficult to design, conduct, and interpret due to the fact that EMFs are imperceptible, ubiquitous, have multiple sources, and can vary greatly over time and short distances” (13). Also, in an animal study, a correlation between ELF-EMF radiation and development of malignant tumors, specifically gliomas and schwannomas of the heart, was discovered (26).

These findings led the American Academy of Pediatrics (AAP) to revise their criteria for EMF exposure in children, and include recommendations such as using hands-free and wired headsets, holding the phone away from the head, limiting television watching, and texting when possible (13). Currently, a 14-country study called MOBI-Kids is being conducted to examine the carcinogenic effects of RF-EMFs from mobile telephones on the central nervous system in children and adolescents (27).

Further upstream, electrosmog has also been shown to induce DNA strand breakages, such that “Any extensive damage or changes to DNA that need repair may increase the risk of developing cancerous cells” (13). Studies also suggest that electrosmog causes genome-wide alterations in methylation (28), or the attachment of one-carbon tags to DNA sequences which modulate gene expression, affecting everything from neurotransmitter production to detoxification.

Mitigating Electrosmog Exposure

Although more data is needed, the science warrants exercising the precautionary principle and taking simple steps to minimize EMF exposure. To remediate electrosmog, renowned doctor Dietrich Klinghardt recommends removing cordless phones from the house, turning off WiFi, switching off fuses at night, considering an EMF-reducing sleep sanctuary or canopy, and grounding.

Moreover, fundamental to neutralizing the toxic effects of electrosmog is spending time in nature and grounding in order to scavenge free radicals and engender antioxidant effects. Direct contact with the surface of the earth precipitates an influx of electrons, which are absorbed and distributed throughout the ground substance of extracellular tissue as well as intracellular biopolymers, neutralizing oxidative stress in the body (29).

Studies have elucidated that grounding decreases the voltage imposed on the body by a factor of seventy upon exposure to alternating current (AC) electric potential (30). This transfer of electrons that occurs as a result of grounding, therefore, can minimize electrosmog-induced derangements in the electrical activities of our bodies, which is meaningful since researchers state that, “There is no question that the body reacts to the presence of environmental electric fields” (30).


References

1. Marshall, T.G., & Heil, T.J.R. (2017). Electrosmog and autoimmune disease. Immunology Research.

2. Curtis, B.D., & Hurtak, J.J. (2004). Consciousness and quantum information processing: Uncovering the foundation for a medicine of light. The Journal of Alternative and Complementary Medicine, 10(1), 27-39.

3. Schwabl, Herbert, and Herbert Klima. “Spontaneous Ultraweak Photon Emission from Biological Systems and the Endogenous Light Field.” Forschende Komplementärmedizin / Research in Complementary Medicine 12, no. 2 (2005): 84-89. doi:10.1159/000083960.

4. Bonilla, E. (2008). [Evidence about the power of intention] [Article in Spanish]. Investigación Clínica 49, 4, 595-615.

5. Hossu, M., & Rupert, R. (2006). Quantum Events of Biophoton Emission Associated with Complementary and Alternative Medicine Therapies: A Descriptive Pilot Study. The Journal of Alternative and Complementary Medicine, 12(2),119-124. doi:10.1089/acm.2006.12.119.

6. Rosch, P.J. (2014). Bioelectromagnetic and Subtle Energy Medicine. Boca Raton: CRC Press.

7. Balmori, A. (2014). Electrosmog and species conservation. Science of the Total Environment, 496, 314-316.

8. Shreder, K. et al. (2016). Low-dose ionising radiation inhibits adipokine induced inflammation in rheumatoid arthritis. Annals of Rheumatological Disease, 75, A64. doi: 10.1136/annrheumdis-2016-209124.151.

9. Lushnikov, K.V. et al. (2001). Effect of extremely high frequency electromagnetic radiation of low intensity on parameters of humoral immunity in healthy mice. Biofizika, 46, 753–760.

10. Mina, D. et al. (2016). Immune responses of a wall lizard to whole-body exposure to radiofrequency electromagnetic radiation. International Journal of Radiation Biology, 92,162–168. doi: 10.3109/09553002.2016.1135262.

11. Gapeev, A.B. et al. (2006). Pharmacological analysis of anti-inflammatory effects of low-intensity extremely high-frequency electromagnetic radiation. Biofizika, 51, 1055–1068.

12. Gapeyev, A.B., Mikhailik, E.N., & Chemeris, N.K. (2008). Anti-inflammatory effects of low-intensity extremely high-frequency electromagnetic radiation: frequency and power dependence. Bioelectromagnetics, 29(3), 197-206.

13. Miah, T., & Kamat, D. (2017). Current understanding of the health effects of electromagnetic fields. Pediatric Annals, 46(4), e172-e174. doi: 10.3928/19382359-20170316-01.

14. Li, Z., & Srivastava, P. (2004). Heat-shock proteins. Current Protocols in Immunology, Appendix 1, Appendix 1 T.

15. Turton, D.A. et al. (2014). Terahertz underdamped vibrational motion governs protein-ligand binding in solution. Nature Communications, 5, 3999. doi: 10.1038/ncomms4999

16. Afzal Mir, M. (1977). Lysozyme: a brief review. Postgraduate Medical Journal, 53, 257-259.

17. Nash, J.A. et al. (2006). The peptidoglycan-degrading property of lysozyme is not required for bactericidal activity in vivo. Journal of Immunology, 177(1), 519-526.

18. Dajani, R. et al. (2005). Lysozyme secretion by submucosal glands protects the airway from bacterial infection. American Journal of Respiratory and Cellular Molecular Biology, 32, 548-552.

19. Bise, W. (1978). Low power radio-frequency and microwave effects on human electroencephalogram and behavior. Physiological Chemistry and Physics, 10(5), 387-398.

20. Zhang, J., Sumich, A., & Wang, G.Y., (2017). Acute effects of radiofrequency electromagnetic field emitted by mobile phone on brain function. Bioelectromagnetics, 38(5), 329-338. doi: 10.1002/bem.22052.

21. Dieudonné, M. (2016). Does electromagnetic hypersensitivity originate from nocebo responses? Indications from a qualitative study. Bioelectromagnetics, 37(1), 14-24.

22. Neustadt, J., & Pieczenik, S.R. (2008). Medication-induced mitochondrial damage and disease. Molecular Nutrition and Food Research, 52, 780-788.

23. Pieczenik, S.R., & Neustadt, J. (2007). Mitochondrial dysfunction and molecular pathways of disease. Experimental and Molecular Pathology, 83, 84-92.

24. Blank, M.M., & Goodman, R. (2009). Electromagnetic fields stress living cells. Pathophysiology, 16(2–3), 71–78. doi:10.1016/j.pathophys.2009.01.006

25. Burlaka, A., Selyuk, M., Gafurov, M., Lukin, S., Potaskalova, V., & Sidorik, E. (2014). Changes in mitochondrial functioning with electromagnetic radiation of ultra high frequency as revealed by electron paramagnetic resonance methods. International Journal of Radiation Biology, 90(5), 357-362.

26. National Toxicology Program, Public Health Services, National Institutes of Health, & US Department of Health and Human Services. (2004). NTP technical report on the toxicology and carcinogenesis studies of Elmiron (Cas No. 37319–17-8) in F344/N rats and B6C3F1 mice (Gavage Studies). National Toxicology Program Technical Report Series, 512, 7–289.

27. Sadetzki, S.S., Langer, C.E., & Bruchim, R. (2014). The MOBI-Kids study protocol: challenges in assessing childhood and adolescent exposure to electromagnetic fields from wireless telecommunication technologies and possible association with brain tumor risk. Frontiers in Public Health, 2, 124. doi:10.3389/fpubh.2014.00124

28. Liu, Y. et al. (2015). Effect of 50 Hz Extremely Low-Frequency Electromagnetic Fields on the DNA Methylation and DNA Methyltransferases in Mouse Spermatocyte-Derived Cell Line GC-2. BioMed Research International.

29. Oschman, J.L. (2009). Charge transfer in the living matrix. Journal of Bodywork and Movement Therapy, 13(3), 215-218.

30. Chevalier, G. et al. (2012). Review article: Earthing: Health Implications of Reconnecting the Human Body to the Earth’s Surface Electrons. Journal of Environmental and Public Health, 1-8.


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Major study reveals processed foods are driving up rates of cancer

processed foods cancer

    
  • Eating processed food significantly raises the risk of cancer, experts have said
  • The disease is claiming more lives because of the popularity of ready meals
  • Sugary cereals and fizzy drinks also dangerous, the wide-ranging study found
  • Families are told to heed the warning and read food labels more carefully

Eating processed food significantly raises the risk of cancer, experts warned last night.

They said the disease was claiming more lives because of the popularity of ready meals, sugary cereals and fizzy drinks.

The products put middle-aged women in particular danger from breast cancer, according to a study in the British Medical Journal.

‘Ultra-processed’ food – any product involving an industrial procedure – now makes up half of our diet.

Packed with chemical additives, the foods bear little resemblance to home-cooked meals. And the more of them an individual eats, the higher their risk of cancer of any type.

Experts believe this is because processed foods, which include packaged meat, pies, sweets and crisps, are higher in fat, salt and sugar.

They also have less of the vitamins and fibre that ward off disease.

The procedures used to make the food – and the chemicals and additives used to boost their flavours and shelf life – are also thought to raise the risk. The researchers said it was the first study to highlight a link between ultra-processed food and an increased overall cancer risk.

Campaigners last night said families should heed the warning and read food labels more carefully to check for levels of fat, salt and sugar.

The research was based on food diaries completed by 105,000 adults. It ranked the participants by how much ultra-processed food they consumed over two 24-hour periods. For those in the top quarter of the sample, 32 per cent of their diet came from ultra-processed food.

They were 23 per cent more likely to develop cancer of any type over the next five years than those in the bottom quarter, whose diet was only 8 per cent ultra-processed food.

Women in the top quarter were 38 per cent more likely to develop postmenopausal breast cancer. The chance of younger women getting premenopausal breast cancer increased 27 per cent, and bowel cancer risk went up 23 per cent. There was no impact on prostate cancer.

pepsi coca cola fizzy drinks

    

Led by experts at the Sorbonne University in Paris, the researchers said Britons were probably even more at risk because they would consume more ultra-processed food than the French adults in the study.

A study of 19 European countries published earlier this month found 50.7 per cent of food sold in the UK is ultra-processed, compared with 46.2 per cent in Germany, 45.9 per cent in Ireland and 14.2 per cent in France. The team assessed 3,300 different food products as part of the study and classed each by the level of processing they had been subjected to.

Sugary products were the most common form of ultra-processed food, making up 26 per cent of foods in this category. Drinks made up 20 per cent and breakfast cereal 16 per cent. The researchers wrote: ‘To our knowledge, this study is the first to investigate and highlight an increase in the risk of overall – and specifically breast – cancer associated with ultra-processed food intake.

‘If confirmed in other populations and settings, these results suggest that the rapidly increasing consumption of ultra-processed foods may drive an increasing burden of cancer in the next decades.’

They found no cancer link to less processed foods – such as canned vegetables, cheeses and freshly made unpackaged bread. And people who mainly ate fresh and unprocessed foods – such as fruit, vegetables, pulses, meat and fish – had a lower cancer risk. Study leader Dr Mathilde Tourier said the poor nutritional value of ultra-processed food was probably the most important factor in cancer risk.

But she added: ‘They all have food additives, they all have compounds formed during the processing and heating of the products, and they have compounds that could come from the packaging itself.

‘Most of the compounds are probably safe, but some of these substances are of concern regarding cancer risk.’

potato chips crisps

    

Tam Fry of the National Obesity Forum said last night: ‘There is no smoke without fire – we should heed the scientists’ fears and read food labels more carefully. Huge quantities of everyday processed food have excessive levels of sugar, fat and salt stuffed in them and it’s all listed on the packaging.

‘Don’t risk cancer by eating anything with, respectively, much more than 15g sugar, 5g saturated fat and 1.5g salt per 100g. That’s not rocket science.’

Carolyn Rogers of the Breast Cancer Care charity added: ‘Now we need to connect the dots and find out if any specific elements in these foods may increase the risk of developing cancer.

‘We know maintaining a healthy weight, through a balanced diet and exercise, is one of the best things you can do for your health. However, lifestyle is one part of a bigger, more complex picture.’

A spokesman for the Food and Drink Federation said: ‘Processed food should not be demonised – by working closely with our partners throughout the food supply chain, we can use processing positively to ensure all sectors of society have access to safe, affordable food.

‘We believe a whole diet and lifestyle approach, which includes consideration of net calorie intake, and not just the role of individual nutrients or ingredients, is the correct way to tackle such issues.’

Dr Alison Tedstone, chief nutritionist at Public Health England, said: ‘A diet high in processed foods is often high in calories, salt, saturated fat and sugar and low in fibre – this increases the risk of some cancers, as does being overweight or obese.

‘Our reduction programmes are making healthier choices easier by reducing the calories, salt and sugar in these types of foods. We’re also helping people to choose healthier options through our Change4Life and OneYou campaigns.’

Source Article from https://www.sott.net/article/377422-Major-study-reveals-processed-foods-are-driving-up-rates-of-cancer

Religious Zionist rabbis call upon Netanyahu to push bill enshrining Torah study as the ‘basic values’ of the Jewish state

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Source Article from https://theuglytruth.wordpress.com/2018/02/13/religious-zionist-rabbis-call-upon-netanyahu-to-push-bill-enshrining-torah-study-as-the-basic-values-of-the-jewish-state/

Zebrafish study: Taking migraine meds while pregnant linked to autism in children


Image: Zebrafish study: Taking migraine meds while pregnant linked to autism in children

(Natural News)
A research study conducted at the Oregon State University and Wenzhou Medical University showed that exposure to a compound used to treat migraines and seizures causes symptoms related to autism. The researchers used zebrafish as a model to conduct a study on autism spectrum disorders (ASD) and their causes.

“The main thing we want to emphasize is the usefulness of this model. We can very quickly look at genetic and environmental factors associated with autism that are very difficult to understand in humans. The ease and speed of the genetic analysis in this model make it very attractive compared to a rodent model,” said co-author Courtney Roper, a postdoctoral fellow in OSU’s College of Agricultural Sciences.

ASD consists of five related neurodevelopmental diseases that are defined by extreme impairments in social interactions, inability to have proper modes of verbal and nonverbal communications, repetitive behavioral patterns, and weird sensitivity to sensory stimulation.

The causes of autism are not known, but it has been linked with both genetic and environmental components, including prenatal exposure to two drugs: thalidomide, a sedative linked to severe birth defects, and valproic acid, which is used to prevent migraine headaches. (Related: Autism, mercury, aluminum and vaccine-induced encephalopathy)

Autism diagnoses have been rising since the 1990s. About one child in 68, will be classified as having autism spectrum disorder, according to the Centers for Disease Control and Prevention (CDC).

ASD affects all ethnic, socioeconomic, and racial groups but is around 4.5 times more common among boys (one in 42) than girls (one in 189).

Zebrafish are small freshwater species that go from a tiny cell to a swimming fish in just five days. They reproduce at a fast rate and are essential for studying the genetic components of vertebrates, including the effects of environmental contaminants and pharmaceuticals on early embryonic development.

Zebrafish have an uncanny likeness to humans at the genetic, cellular, and molecular levels; this means that the implications of findings associated with zebrafish are extremely relevant to humans.

Studies on embryonic zebrafish are particularly important, for embryonic zebrafish develop quickly, are transparent, and can easily thrive even in small amounts of water.

The research’s findings concluded that treating zebrafish embryos with valproic acid starting eight hours after fertilization results in a significant rise in the ASD macrocephalic phenotype, that is, an enlarged head.

It also caused hyperactivity in embryos and larvae.

“In addition to the ASD-like characteristics, we observed an overgrowth of mature newborn neurons and neural stem cells in the developing brain after VPA exposure. Next, now that we have models for behavior established, is to start looking at the genetic and environmental interactions, examining some of the potential factors that might be driving autism in humans,” Roper said.

The findings were published in the journal Neurotoxicology and Teratology. The National Natural Science Foundation of China supported the conduct of this research.

More on autism spectrum disorder

Some of the behavioral and medical conditions that are associated with autism spectrum disorder include anxiety, attention deficit hyperactivity disorder, gastrointestinal problems, depression, epilepsy, and sleep disturbances.

According to the CDC, children with autism are:

  • 1.8 times more likely than children without developmental illnesses to have asthma
  • 1.8 times more likely to have eczema
  • 1.8 times more likely to have food allergies
  • 2.2 times more likely to have chronic headaches
  • 3.5 times more likely to have chronic diarrhea

For more stories on disorders of the mind and behavior, visit Mind.news.

 

Sources include:

Science.news

ScienceDaily.com

AutismSpeaks.org

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Source Article from http://www.naturalnews.com/2018-02-11-zebrafish-study-taking-migraine-meds-while-pregnant-linked-to-autism-in-children.html

Israeli law enforcement woefully lax, says study by Chief Economist

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Source Article from https://theuglytruth.wordpress.com/2018/02/09/israeli-law-enforcement-woefully-lax-says-study-by-chief-economist/

Study finds BPA in 86% of teenagers

And that was after one week of avoiding foods that may have come into contact with the notorious hormone-disrupting chemical!

A recent study from the University of Exeter has found traces of bisphenol A (BPA) in 86 percent of teenagers. This is concerning, since BPA is a known hormone-disrupting chemical that imitates female sex hormones and has been linked to breast and prostate cancers, as well as low sperm counts and sperm disfigurements in men.

Despite its bad reputation, BPA continues to be used in many plastic containers, water bottles, food cans, dental floss, and heat-resistant papers, which means that humans come into contact with it frequently.

This particular study set out to see if it was possible to reduce one’s BPA level by altering dietary choices. It was designed to be a ‘real-world setting’, unlike prior studies that have focused on families and related individuals, who likely share sources of BPA, and participated in strict dietary interventions that are not realistically sustainable. From the discussion:

“Our intervention is a ‘real-world’ diet, designed to a set of guidelines (such as reduction in the usage of tinned foods or foods with high levels of processing), rather than the strict, prescribed diets that have been used in other studies, which suggested that it was possible for participants to reduce their urinary BPA excretion by approximately 60% in a period of just 3 days. In our self-designed, self-administered study this was unachievable.”

Participants included 94 students between the ages of 17 and 19 from schools in southwestern England. They followed a BPA-reduction diet for seven days. This included switching to stainless steel and glass food containers, not microwaving food in plastic, washing their hands after handling receipts, avoiding canned foods and takeout in plastic, and using a coffee filter or percolator instead of plastic coffee makers that may contain polycarbonate-based water tanks and phthalate-based tubing. The students gave urine samples before and after the interventions.

The conclusion?

“Participants were unable to achieve a reduction in their urinary BPA over the 7-day trial period, despite good compliance to supplied guidelines.”

This alarming discovery goes to show that BPA is so ubiquitous in our environment that, even when we take measures to minimize exposure, it’s impossible to avoid entirely. Where it’s coming from, however, is unclear. The study authors write that exposure can happen through dust ingestion and skin absorption, and that BPA can leach into food from polycarbonate or epoxy resins after manufacture. The migration rate increases with higher temperatures, and with time and use (which is why you should never reuse a disposable plastic water bottle or microwave food in plastic).

The majority of study participants (66 percent) said it would be difficult to maintain the BPA-reduction diet over the long term, due to inconsistent labelling, sourcing challenges, and having to alter food preferences. Comments included:

“Almost everything is packaged in plastic.” “The biggest problem was that a lot of packaging doesn’t state what type of plastic it is or whether it contains BPA.” “You can’t get it all from supermarkets.” “[I] had to go to more individual food shops”.”

The researchers are calling for more consistent labelling on packaging to make it easier for people to avoid BPA. As Professor Lorna Harries, one of the study authors, told the University of Exeter:

“In an ideal world, we would have a choice over what we put into our bodies. At the present time, since it is difficult to identify which foods and packaging contain BPA, it is not possible to make that choice.”

Source Article from https://www.treehugger.com/health/study-found-bpa-86-teenagers.html

New Study Links Human Consciousness To A Law That Governs The Universe

By Chelsea Gohd, Futurism

Human Entropy

Our species has long agonized over the concept of human consciousness. What exactly causes it, and why did we evolve to experience consciousness? Now, a new study has uncovered a clue in the hunt for answers, and it reveals that the human brain might have more in common with the universe than we could have imagined.

According to a team of researchers from France and Canada, our brains might produce consciousness as something of a side effect of increasing entropy, a process that has been taking place throughout the universe since the Big Bang.

Their study has been accepted for publication in the journal Physical Review E.

The concept of entropy is famously confusing, and the definition has evolved over time. Essentially, entropy is a thermodynamic property that refers to the degree of disorder or randomness in a system. It can be summed up as the description of a system’s progression from order to disorder.

The second law of thermodynamics states that entropy can only remain constant or increase within a closed system — a system cannot move from high entropy to low entropy without outside interference. A common example that demonstrates entropy is an ice cube melting — the cube is in a state of low entropy, but as it melts and disorder grows, entropy increases.

Many physicists think that the universe itself is in a constant state of increasing entropy. When the Big Bang occurred, the universe was in a state of low entropy, and as it continues to gradually spread out, it is growing into a higher entropy system. Based on this new study, our brain may be undergoing something similar, and consciousness happens to be a side effect of the process.

The Brain and Disorder

To see how the concept of entropy could be applied to the human brain, the researchers analyzed the amount of order in our brains while we’re conscious compared to when we’re not. They did this by modeling the networks of neurons in the brains of nine participants, seven of whom had epilepsy.

They looked at whether or not neurons were oscillating in phase with one another as this could tell them if the brain cells were linked. They compared observations from when patients were awake, when they were asleep, and when patients with epilepsy were having seizures.

The researchers found that the participants’ brains displayed higher entropy when fully conscious. “We find a surprisingly simple result: normal wakeful states are characterized by the greatest number of possible configurations of interactions between brain networks, representing highest entropy values,” the team wrote in the study.

This finding prompted the researchers to suggest that consciousness might be a side effect of a system working to maximize information exchange. In other words, human consciousness emerges due to increasing entropy.

While the team’s theory is exciting and will likely lead to further research exploring a potential link between human consciousness and entropy, it is far from conclusive. The study’s sample size was exceptionally small, so they’ll need to replicate their results on larger groups and different types of brain states. Still, it provides a fascinating explanation for human consciousness and may be the clue that eventually helps us fully understand the strange phenomenon.

Image Credit: Wikimedia 

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Source Article from https://truththeory.com/2018/02/05/new-study-links-human-consciousness-law-governs-universe/